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To better show its efficacy, we included a control group that was treated with rosiglitazone, a type of lel TZD. Abstract Ginkgo biloba, an herbal medication, is capable of lowering glucose, fat, and lipid peroxide in diabetic patients.
Oei treatment also increased the number of trabecular bones in DEX-induced rats. Abstract Our aim is to investigate the potential therapeutic value of morin against osteoporosis and elucidate the lsi of action. Journal of Natural Medicines [03 Sep65 1: It is said that GBE treatment could avoid drug-induced obesity.
Furthermore, after analyzing gene expression, we suggest that GBE chiefly exerts its effects by stimulating IRS-2 transcription. Or filter your current 14118. The relative expression of osteogenic and bone resorption markers was determined by real-time polymerase chain reaction and Western blotting, respectively.
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CitePeer Related Articles http: We investigated the effects of GBE on glucose consumption, glucokinase activity, and mRNA levels of key genes in glucose metabolism and the insulin signaling pathway. Mechanistically, morin reversed the decrease of osteogenic markers and increase of bone resorption markers, which might 114118 be mediated by modulation of MAPK signaling cascades.
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Pathological examination was performed by hematoxylin and eosin staining. Our data suggest that GBE has the potential to prevent insulin resistance and is a promising anti-diabetic drug. Gene Ontology GO Terms. Find all citations in this journal default. The data indicated that GBE repressed glucose uptake under normal conditions, while it dramatically improved glucose tolerance under insulin-resistant conditions.
It should be noted that, unlike rosiglitazone, GBE did not stimulate excessive glucose uptake as it improved glucose tolerance.
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Body weight and BMD were both significantly decreased in osteoporotic rats, although BMD was partially restored by intraperitoneal morin administration.